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by
Thai, Kevin K., author.
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contribute to uncontrolled cell divisions. Generally, cancer cells have acquired gain of function mutations
by
Choi, Jaewoo, author.
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cancer. Despite the significant progress, much more remains to be explored in the field of ubiquitin and
by
West, Kirk Leon, author.
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each be further interrogated. The SWI/SNF and SAGA complexes have been implicated in cancer and may
by
Flores, Aimee Alyssa, author.
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adult stem cell. One such cancer is squamous cell carcinoma, for which recent studies in our lab traced
by
Runge, John Stevens, author.
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misregulation is a hallmark of cancer where endogenous regulatory programs are co-opted to promote aberrant
by
Wolfson, Benjamin, author.
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chondrocyte differentiation, and we have shown that miR-140 is a tumor suppressor miRNA that inhibits cancer
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Thurston, Alison K., author.
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sequence is generally thought of as harmful, as it is linked with many forms of cancer and hereditary
by
Antonia, Ricardo J., author.
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directly phosphorylates Thr172 within the AMPK activation loop, both basally in cancer cell lines and in
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Nelson, Adam, author.
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proteins in colon cancer HCT116 cells, showed that L164P, and not L164A/T165A, significantly impaired
by
Pulliam, Nicholas, author.
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-dependent manner, ovarian and breast cancer cells to PARPi. In addition, DNMTi enhanced PARP 'trapping' by
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Fisher, Timothy B., author.
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Background and Significance: Epithelial ovarian cancer (EOC) is the leading cause of cancer death
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Duchemin, Nicholas Joseph, author.
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leading cause of primary liver cancer. Understanding the cellular pathways that contribute to the control

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