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by
Su, Huizhong, author.
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Metabolic basis of myc-induced apoptosis / Su, Huizhong, author.
by
Xu, Yiyue, author.
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Reversal of Apoptosis and Cancer Development / Xu, Yiyue, author.
by
Erler, Janine, author.
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Hypoxia and apoptosis in colon cancer / Erler, Janine, author.
by
Liu, Qiang, author.
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Autophagy and Apoptosis in Acute Myeloid Leukemia / Liu, Qiang, author.
by
McDonnell, S. J., author.
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Study of novel protein-protein interactions modulating perp-mediated apoptosis / McDonnell, S. J
by
Ethaeb, Ali Mahdi, author.
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Elucidation of the signalling mechanisms involved in tf-mediated apoptosis in endothelial cells /
by
Yu, Chen, author.
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G9a, Glp and HP1gamma Facilitate Glucocorticoid-Induced Apoptosis in B-ALL Cells / Yu, Chen, author.
by
Patience, Lauren Alexandra, author.
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Apoptosis-driven microenvironmental conditioning by microvesicles in non-hodgkin lymphoma /
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Chu, Ying Ying Julia, author.
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Apoptosis is promoted by unconventional fcγr-pi3kcdc42-pak-mek-erk signalling in the human
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Felton, Jennifer Marie, author.
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Investigation of the role of mcl-1 and mer in the regulation of eosinophil apoptosis and
by
Sun, Jinquan, author.
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A Novel Small Molecule DNA Intercalator PZ1225 in Cancer Cell Apoptosis and a Histone Modifier PAD4
by
Nguyen, Jeffrey, author.
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Investigating the Function and Regulation of Prostate Apoptosis Response-4 in Cancer / Nguyen
by
Zhao, Xiaofeng, author.
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Can promotion of neutrophil apoptosis enhance repair in the infarcted myocardium and resolution of
by
Lynch, James Thomas, author.
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Molecular Mechanisms Conferring Resistance/Sensitivity to Glucocorticoid-Induced Apoptosis during
by
Kwok, Ellius, author.
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CDKL5 iPSC-derived Neural Cells Exhibit Deficiencies in Growth and Increased Apoptosis / Kwok
by
Uzoegwu, Chukwumamkpam Chukwudera, author.
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Survival and Apoptosis / Uzoegwu, Chukwumamkpam Chukwudera, author.
by
Vaidya, Mayuri Manoj, author. (orcid)0000-0001-7152-5558
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Verification of Apoptosis in MDA-MB-231 Triple Negative Breast Cancer Cells Post NBA Photodynamic
by
Qian, Jiang, author.
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Characterization of Transcription-independent APC Tumor Suppressor Function in Apoptosis / Qian
by
Irvine, Jane, author.
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Investigation of apoptosis in T-cell lymphomas of mice carrying a c-Myc transgene / Irvine, Jane
by
Gill, Jason Harry, author.
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Suppression of hepatocyte apoptosis by non-genotoxic carcinogens / Gill, Jason Harry, author.
by
Merritt, Anita Jane, author.
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The role of bcl-2 and p53 in the regulation of apoptosis in the gastrointestinal tract / Merritt
by
Kanuri, Sri Harsha, author.
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Role of Spleen Tyrosine Kinase in Tumor Necrosis Factor Like Weak Inhibitor of Apoptosis Induced
by
Evans, Dyfed Llyr, author.
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The induction of apoptosis by the anticancer agent cisplatin / Evans, Dyfed Llyr, author.
by
Gurol, Kerem Cahit, author.
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associated with a myriad of cellular pathways, including apoptosis. However, the mechanism and function of
by
Yang, Xi, author.
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, Annexin-V apoptosis detection and tumor growth in vitro, have been demonstrated with statistical
by
Branco, Patrícia Alexandra Batista, author.
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H+-influx/-efflux. They also induce cell molecular markers typical of death by apoptosis in H
by
Skorupa, Jennifer A., author.
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Ribotoxic stressors such as anisomycin (ANS) and deoxynivalenol (DON) induce apoptosis in MAC
by
Wang, Yihan, author.
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) significantly impaired H-HAECs tube formation and proliferation, and induced cell apoptosis, all of which were
by
Cantwell, Marc Taylor, author.
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differentiation stage. Without STAT3, the brown preadipocytes have increased apoptosis early in the terminal
by
Jeffus, Dana A., author.
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apoptosis resistance, potentially leading to carcinogenesis. Previous research shows that under rapamycin
by
Ruff, Amanda, author.
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expression in breast cancer cells and effectively promote apoptosis in estrogen receptor negative breast
by
Naidu, Chitra Upendra, author.
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and apoptosis. Overexpression of mammalian PLC-gamma is linked to tumorigenesis and the transition of
by
Jiao, Meng, author.
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migration and apoptosis. The life of a bleb cycle includes bleb initiation, expansion, and retraction. While
by
Ponder, Jessica Nicole, author.
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the mechanism by which galectin-1 protects leukemic stem cells from apoptosis and how these compounds
by
Ramirez, Mireya Nael, author.
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cancer cells. Erastin-induced ferroptosis differs from apoptosis, necrosis and autophagy, since it leads
by
Alkandari, Abdulwahab, author.
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results in cytotoxicity, apoptosis, and DNA damage. Conclusions: In-vitro studies demonstrated that ENDS
by
Liu, James Y., author.
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induces the cancer cells to undergo apoptosis (programmed cell death). We find a reduction in 15B cell
by
Roberts, Morgan C., author.
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depleted uranium, in the form of uranyl acetate (UA) by looking at DNA damage repair and cellular apoptosis
by
Oben, Karine Z., author.
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)-activated JNK/AP-1 signaling is a major pathway mediating apoptosis of MDS-L cells by WFA. Increase in ROS
by
Paskins, Aimee Rebecca, author.
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intracellular aggregates, an increase in apoptosis, and decreased cell viability. ESI-IMS-MS demonstrated
by
Hu, Wenbao, author.
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-linked inhibitor of apoptosis). XIAP is also a RING-domain E3 ubiquitin ligase of multiple signaling proteins
by
Alonzo, Judith Raquel, author. (orcid)0000-0003-0603-9187
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and eventually apoptosis. Our knowledge of factors that contribute to uracil accumulation in mtDNA is
by
Schultz, Laura Elizabeth, author.
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homozygotes transiently suppressed neural progenitor apoptosis, indicating the absence of Rbbp4 activates TP53
by
Magani, Fiorella, author.
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proliferation, increased apoptosis, and decreased migration. Second, as an indirect approach to target cells
by
Gill, Taylor Elizabeth, author.
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expression programs responsible for cellular proliferation, growth, metabolism, and evasion from apoptosis
by
Elliott, Eric Isaac, author.
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responses by forming inflammasomes with the adaptor protein apoptosis-associated speck like protein with a
by
Okumu, Denis O., author.
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-regulated the expression and stability of BIRC6, a member of the inhibitor of apoptosis proteins (IAP) family
by
Blake, Courtney Renee, author.
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/mL) on H1299 proliferation were determined 24 hrs. or 48 hrs. (cytotoxic/apoptosis) after plating
by
Hurtado, Myrna, author.
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popularity for its anti-cancer properties such as inhibition of cell growth and induction of apoptosis in
by
Laing, Brenton, author.
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hypothalamic leptin sensitivity and reduced pro-opiomelanocortin neuronal apoptosis.
by
Guo, Rui, author.
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mitochondrion was unable to rescue infected cells from apoptosis/necrosis. Certain PRRSV proteins were detected
by
Scott, Celeste, author.
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-carcinogenic properties through apoptosis and induction of autophagy in several human cancer cell lines. Apigenin has
by
Uddin, Mohammad Burhan, author.
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R273H. In addition to dysfunction in proliferation arrest and apoptosis, our further study indicates
by
Griesenauer, Brad, author.
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type 1 response without affecting T cell proliferation, apoptosis, or migration. In our aGVHD models
by
Rowell, Temperance Rebecca, author.
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intracellular Ca2+ signaling, which can affect cell proliferation and apoptosis. Specifically, we demonstrated
by
Lerman, Irina, author.
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cell lines. Knockdown of SERPINB1 in RWPE-1 cells increases proliferation, decreases apoptosis, and
by
Wallace, LaShanale, author.
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regulate different pathways involved in oxidative phosphorylation, apoptosis, and oxidative stress, all of
by
Stuart, Johnasha Donna, author.
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death gene array analysis revealed that T1 infection induced expression of genes involved in apoptosis
by
Aslamy, Arianne, author.
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-induced apoptosis and reduces ER stress; this is dependent on C2AB's calcium binding capacity. C2AB is sufficient to
by
Affronti, Hayley Catherine, author. (orcid)0000-0001-9751-0629
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blocks CaP growth in vitro and in vivo, and induces apoptosis in human samples tested in an ex vivo
by
Fan, Aaron E., author.
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increasing CD8 T cell persistence and resistance to apoptosis. Here, I aimed to test the hypothesis that CA
by
Ahmed, Roshan, author.
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fatty acid synthase (FASN) inhibition using novel small molecule inhibitor IPI-9119 induces apoptosis in
by
Bacigalupa, Zachary A., author.
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tissue and isolated epithelial organoids contained reduced MUC2 and elevated apoptosis suggesting a
by
Azab, Ehab, author.
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-GsalphaCtrl and non-treated cells. Osteoclast apoptosis was significantly enhanced by Ocy-GsalphaCtrl and Ocy
by
Bjersand, Kathrine, author.
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study I showed that expression of Napsin A was associated with expression of the apoptosis regulators p
by
McCutcheon, Sean, author.
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apoptosis as a chemical trigger for bone remodeling, the Mmicron system will further elucidate bone function
by
Volakis, Leonithas Ioannis, author.
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events of apoptosis in lung cancer cells.
by
Khar, Karen, author.
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drug for patients with this type of destabilized p53 mutation could lead to apoptosis in cancer cells.
by
Johnson, Zariel I., author.
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matrix catabolism, cellular senescence, and apoptosis play an important role. In immune and other cell
by
Ren, Jun, author.
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injury with apoptosis, loss of contractile phenotype as well as regain of proliferative and migratory
by
Cao, Xiaolong, author.
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-JNK-p38, JAK-STAT, autophagy, apoptosis and RNA interference pathways, analyzed their evolution and mRNA
by
Zhang, Leilei, author.
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CC3 are selected as cell death model to further investigate the cell apoptosis after injection. The
by
Ye, Weiping, author.
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(-)-GPCSO induced apoptosis in several human breast cancer cells. It induced DNA fragmentation in
by
Jeffries, Shawn, author.
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-fate decisions such as differentiation, proliferation and apoptosis through direct intercellular contact
by
Bibi, Rufzan Rushna, author.
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function of E6 can induce the apoptosis of cultured HPV-16 positive cervical carcinoma cells. Our intention
by
Issa, Yaser, author.
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dependent fashion. Biochemical and morphological features of apoptosis were observed in HgCl2 induced death
by
Vickers, Elaine R., author.
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apoptosis. Thus, SRF-dependent Elk-1 activity might play a role in cell survival. In order to identify Elk-1
by
Caneus, Julbert, author.
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relative to negative controls. Our data also showed significantly increased levels of apoptosis in
by
Cheng, Lixin, author.
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connectivity, compared with the apoptosis proteins; and (4) the proteins targeted by small-molecule drugs show
by
Zhang, Li, author.
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expression of apoptosis markers cleaved caspase-9,-7,-3 and PARP and cell cycle inhibitors p53, p21 and p27
by
Bastola, Prabhakar, author.
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apoptosis. Mechanistic studies point to the unresolved unfolded protein response as a mechanism by which VCP
by
Withers, Henry G., author. (orcid)0000-0002-9746-9352
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be critical nodes regulating adhesion, survival, and apoptosis pathways and included death receptors
by
Garcia, Luis Daniel Borrero, author.
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gefitinib, suggesting bypass mechanisms of therapy resistance. Lapatinib is known to promote apoptosis in
by
Glassman, Fiona Yau, author.
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, especially in hemostasis and apoptosis. The exposure of PS on the surface of apoptotic cells functions as an
by
Weeramange, Chamitha Janani, author.
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formation of a deoxy-6-thioguanosine triphosphate mimic that is incorporated into DNA resulting in apoptosis
by
Islam, Shariful, author.
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-xL results in induced apoptosis evident by PARP cleavage. In a DE-DLBCL SCID mouse xenograft model, ibrutinib
by
Zhang, Zishan, author.
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apoptosis. These studies suggest that UPR activation might be enhanced in CFTR KO cells, but the specific
by
Putzbach, William, author. (orcid)0000-0002-2669-6654
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-length apoptosis-inducing proteins are toxic to cells, even those harboring homozygous deletion of the CD95 gene
by
Jamali, Nasim, author.
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demonstrated that calcitriol decreased proliferation and migration of retinal PC in culture. Although apoptosis
by
Nkembo, Augustine T., author.
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examined. PCAIs induced apoptosis with EC 50 values ranging from 2.4 to 5.1, 2.0 to 7.3, 1.8 to 3.3 and 7.0
by
Poku, Rosemary A., author.
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-cancer effects in mCRPC cells, at least in part through the induction of caspase-mediated apoptosis and
by
Hussein, Hayam, author.
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unmethylated CpG (5 mug/ml). Cathepsin K inhibition promoted BMNC viability and reduced cell apoptosis
by
Craggs, Graham, author.
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functional complementation within the gene family and promotion of apoptosis.
by
Robertson, Naomi, author.
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lines regarding induction of apoptosis after mitomycin C exposure. In conclusion, this study formed part
by
O'Farrell, Anne-Marie, author.
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however, delay the onset of apoptosis in this cell line. FDCP-Mix A4 cells therefore provide evidence of a
by
Bhatia, Hemlata, author.
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the supplementation of zinc. Genes from various pathways were observed to be affected: apoptosis
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