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Subject: Oncology.
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by
Mallaney, Cates N., author.
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Kdm6b is Required for Self-Renewal of Normal and Leukemic Mouse Stem Cells Under Proliferative
by
Quesenberry, Peter J. editor.
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Cancer Stem Cells: A Matter of Perspective -- B and T-cell development -- HIV lymphomagenesis
by
Ponder, Jessica Nicole, author.
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leukemia (AML). Using the M9-ENL cell line as a high-throughput screening model for leukemic stem cells
by
Liu, Xuxiang, author.
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facilitate the growth of cancer cells in bone. High expression of miR-218 in breast cancer cells increases
by
Bartlebaugh, Jordan Michael Elizabeth, author.
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-specific transcription factors. We show that loss of Phf6 in B-cell leukemia results in systematic changes in gene
by
Fang, Chun-Sheng, author.
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optimization cycles based on the modifications of ring A, ring B, and linker C of compound 1, which generated
by
Ratnam, Nivedita Mohan, author.
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. Mechanistically, we found that GDF-15 suppresses macrophage activity by inhibiting TGF-b activated kinase (TAK1
by
Foss, Francine. editor.
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to B-cell lymphomas in the Asian basin. While the overall incidence of B-cell lymphomas has begun to
by
Kirkpatrick, Gregory D., author.
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early hematopoietic precursors or myeloid lineage cells, but can then later be converted to a B cell
by
Vande Woude, George F.
ScienceDirect http://www.sciencedirect.com/science/book/9780120066971
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Cell Transplantation for Solid Cancers; VII. Mesenchymal Stem Cells; A. Surface Markers and Homing; B
by
Ewing, Joanne, author.
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allowing development of tolerance in the absence of GVHD. Using C57B1/6 (H-2b) as recipients of Balb/c (H-2
by
Chiu, Yi-Lin, author. (orcid)0000-0002-1827-6924
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proteolysis medicated by co-expression of HAI-2 contributes to various malignant aspects of neoplastic B-cells
by
Gomes, Cecil, author.
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nuclear-reporter H2B-GFP, we were able to show that these polyploid cells are capable of entering mitosis
by
Hartley, Genevieve, author.
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B cells, but low to negative expression of PD-1 and PD-L1 by both healthy and malignant T cells
by
Islam, Shariful, author.
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polyploid cells (TIP). These TIP cells exhibited a high metabolic rate by increased AKT/mTOR and ERK/MAPK
by
Plebanek, Michael Paul, author.
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cells that express scavenger receptor-type B1 (SR-B1). By modulating cholesterol flux at lipid rafts
by
Poku, Rosemary A., author.
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interactions as a means to mitigate excessive growth signaling in cancer cells. We investigated the effects of
by
Robertson, Naomi, author.
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sensitivity of these cell lines to this drug. The MTT and sulforhodamine B (SRB) assays were susceptible to
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